UP - logo

Rezultati iskanja

Osnovno iskanje    Izbirno iskanje   
Iskalna
zahteva
Knjižnica

Trenutno NISTE avtorizirani za dostop do e-virov UPUK. Za polni dostop se PRIJAVITE.

6 7 8 9 10
zadetkov: 93
71.
  • The Raf-MEK-ERK Cascade Rep... The Raf-MEK-ERK Cascade Represents a Common Pathway for Alteration of Intracellular Calcium by Ras and Protein Kinase C in Cardiac Myocytes
    Ho, Peter D; Zechner, Dietmar K; He, Huaping ... The Journal of biological chemistry, 08/1998, Letnik: 273, Številka: 34
    Journal Article
    Recenzirano
    Odprti dostop

    Ras and protein kinase C (PKC), which regulate the Raf-MEK-ERK cascade, may participate in the development of cardiac hypertrophy, a condition characterized by diminished and prolonged contractile ...
Celotno besedilo
72.
  • Pro-inflammatory Cytokines ... Pro-inflammatory Cytokines Stimulate Mitogen-activated Protein Kinase Subfamilies, Increase Phosphorylation of c-Jun and ATF2 and Upregulate c-Jun Protein in Neonatal Rat Ventricular Myocytes
    Clerk, Angela; Harrison, Joanne G; Long, Carlin S ... Journal of molecular and cellular cardiology, December 1999, 1999-Dec, 1999-12-00, 19991201, Letnik: 31, Številka: 12
    Journal Article
    Recenzirano

    A. Clerk, J. G. Harrison, C. S. Long and P. H. Sugden. Pro-inflammatory Cytokines Stimulate Mitogen-activated Protein Kinase Subfamilies, Increase Phosphorylation of c-Jun and ATF2 and Upregulate ...
Celotno besedilo
73.
  • c-Jun NH2-terminal Kinase A... c-Jun NH2-terminal Kinase Activation Leads to a FADD-dependent but Fas Ligand-independent Cell Death in Jurkat T Cells
    Chen, Yun; Lai, Ming-Zong The Journal of biological chemistry, March 2001, 20010316, 2001-Mar-16, 2001-03-00, Letnik: 276, Številka: 11
    Journal Article
    Recenzirano
    Odprti dostop

    Persistent c-Jun NH2-terminal kinase (JNK) activation induces cell death. Different mechanisms are ascribed to JNK-induced cell death. Most of the JNK-apoptosis studies employ stress stimuli known to ...
Celotno besedilo
74.
  • Angiotensin Blockade Inhibi... Angiotensin Blockade Inhibits Increased JNKs, AP-1 and NF- κ B DNA-binding Activities in Myocardial Infarcted Rats
    Yoshiyama, Minoru; Omura, Takashi; Takeuchi, Kazuhide ... Journal of molecular and cellular cardiology, April 2001, 2001-Apr, 2001-04-00, 20010401, Letnik: 33, Številka: 4
    Journal Article
    Recenzirano

    Inhibition of the renin–angiotensin system has been shown to prevent left ventricular remodeling after myocardial infarction. However, the effect of angiotensin on the signal transduction pathway of ...
Celotno besedilo
75.
  • Overexpression of 12-Lipoxy... Overexpression of 12-Lipoxygenase and Cardiac Fibroblast Hypertrophy
    Wen, Yeshao; Gu, Jiali; Peng, Xianghong ... Trends in Cardiovascular Medicine, 2003, 2003-May, 2003-05-00, 20030501, Letnik: 13, Številka: 4
    Book Review, Journal Article
    Recenzirano

    Leukocyte-type 12-lipoxygenase (12-LO) catalyzes the conversion of arachidonic acid (C20:4) to 12-hydroperoxyeicosatetraenoic acid, which in turn reduces to 12-hydroxyeicosatetraenoic acid (12-HETE) ...
Celotno besedilo
76.
  • Antioxidant Therapy Attenua... Antioxidant Therapy Attenuates JNK Activation and Apoptosis in the Remote Noninfarcted Myocardium after Large Myocardial Infarction
    Li, Wei-Gen; Coppey, Lawrence; Weiss, Robert M ... Biochemical and biophysical research communications, 01/2001, Letnik: 280, Številka: 1
    Journal Article
    Recenzirano

    We hypothesized that the concomitant occurrence of increased oxidative stress, JNK activation, and myocyte apoptosis in the remote myocardium (RM) following a large myocardial infarction (MI) are ...
Celotno besedilo
77.
  • Signal transduction in card... Signal transduction in cardiac hypertrophy — dissecting compensatory versus pathological pathways utilizing a transgenic approach
    Wang, Yibin Current Opinion in Pharmacology, 2001, 2001-Apr, 2001-04-01, 20010401, Letnik: 1, Številka: 2
    Book Review, Journal Article
    Recenzirano

    Targeted and regulated genetic manipulation, physiological intervention to introduce biomechanical stress and injury, sophisticated measurement of cardiac function in transgenic heart at whole organ ...
Celotno besedilo
78.
  • Propranolol prevents enhanc... Propranolol prevents enhanced stress signaling in Gsα cardiomyopathy: potential mechanism for β-blockade in heart failure
    Karoor, Vijaya; Vatner, Stephen F; Takagi, Gen ... Journal of molecular and cellular cardiology, 2004, 2004-Feb, 2004-02-00, 20040201, Letnik: 36, Številka: 2
    Journal Article
    Recenzirano

    Beta-adrenergic receptor (β-AR) blockade is now widely utilized therapeutically for heart failure, but its cellular mechanism of action is not clear. Mice with cardiac-specific overexpressed Gsα ...
Celotno besedilo
79.
  • Tissue Distribution and Fun... Tissue Distribution and Functional Expression of a cDNA Encoding a Novel Mixed Lineage Kinase
    Bloem, Laura J; Pickard, Todd R; Acton, Susan ... Journal of molecular and cellular cardiology, September 2001, 2001-Sep, 2001-09-00, 20010901, Letnik: 33, Številka: 9
    Journal Article
    Recenzirano

    Hypertrophy is an adaptive response of the heart to myocardial injury or hemodynamic overload that may progress and contribute to cardiac decompensation and eventually to heart failure. The signaling ...
Celotno besedilo
80.
  • Calcineurin inhibitors and ... Calcineurin inhibitors and cardiac hypertrophy
    Force, Thomas; Rosenzweig, Anthony; Choukroun, Gabriel ... The Lancet (British edition), 1999, 1999-Apr-17, 1999-04-00, 19990417, Letnik: 353, Številka: 9161
    Journal Article
    Recenzirano

    Research on the effects of cyclosporin on the progression of heart enlargement indicates that the drug alone cannot prevent cardiac hypertrophy, but may block one physiological pathway of its ...
Celotno besedilo
6 7 8 9 10
zadetkov: 93

Nalaganje filtrov